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Fig. 3 | BMC Complementary and Alternative Medicine

Fig. 3

From: Bioactive components and mechanisms of Chinese poplar propolis alleviates oxidized low-density lipoprotein-induced endothelial cells injury

Fig. 3

EACP inhibited the levels of caspase 3, PARP cleavage, the ratio of LC3-II/LC3-I and enhanced the p62 level in ox-LDL induced HUVECs at 6 h. a, Expression of procaspase 3 and full length PARP after treatment with EACP in ox-LDL induced HUVECs; b, Quantification of relative expression quantity in ox-LDL induced HUVECs at 6 h; c, Cells were stained with anti-LC3B antibody for immunostaining. Immunofluorescence graphs showed a decrease of endogenous punctuate LC3 after treatment with EACP; d, Expression of p62 and LC3B in ox-LDL induced HUVECs; e, Quantification of relative expression quantity in ox-LDL induced HUVECs at 6 h, respectively. (*P < 0.05, **P < 0.01 vs ox-LDL group, n = 3). Data are means ± S.E.M.

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BMC Complementary Medicine and Therapies

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