Fig. 8From: MOK, a pharmacopuncture medicine, regulates thyroid dysfunction in L-thyroxin-induced hyperthyroidism in rats through the regulation of oxidation and the TRPV1 ion channelEffect of MOK pharmacopuncture on the oxidation in liver, brain, and heart tissues ofLT4-induced hyperthyroidism rats. MOK pharmacopuncture was subcutaneously administration once daily for 2 weeks, and measured the levels of GSH (a) from the liver of rats by ELISA. Theexpression of catalase (CAT) and SOD2in the liver (b), brain (c), and heart (d) tissues using Western blot. Data are presented as mean ± SD (n = 5 per each group). * P < 0.05, ** P < 0.01 and *** P < 0.001 vs normal (a) or control (b). Normal, normal group; Control, LT4-induced hyperthyroidism group; MOK 0.3, MOK 0.3 mg/kg-treated group in control; MOK 3, MOK 3 mg/kg-treated group in control; and PTU, PTU 10 mg/kg-treated group as a reference drugBack to article page