Figure 4From: 1’-Acetoxychavicol acetate inhibits growth of human oral carcinoma xenograft in mice and potentiates cisplatin effect via proinflammatory microenvironment alterations ACA reduces NF-κB activation in HSC-4 human oral cancer cells. (A) Reduction in IKK-α/β key phosphorylation sites prevents subsequent phosphorylation of IκB-α at Ser32/36, resulting in NF-κB inactivation. (B) All band intensities were quantified and normalized against β-actin using the ImageJ v1.43u software, and presented as mean ± SEM of three replicates.Back to article page